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[A functional blockade of Wnt/?-catenin pathway by either a pharmacological Wnt-antagonist, WntC59, sulidac sulfide, or ?-catenin (functional read out of Wnt/?-catenin pathway) SiRNA mediated genetic manipulation demonstrated that a functional perturbation of the pathway is causal to the metastasis- associated phenotypes including fibronectin-directed migration, F-actin organization, and invasion in TNBC cells.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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